ischemia reperfusion

The role of apoptosis and Fas/ FasL system in rabbits with lung ischemia reperfusion injury

细胞凋亡和Fas/FasL系统在兔肺缺血再灌注损伤中的作用

The destruction of BBB is an important mechanism of cerebral ischemia-reperfusion injury.

血脑屏障破坏是脑缺血再灌注(ischemiareperfusion,IR) 脑损伤的重要的病理生理基础.

CONCLUSION Patients surffer gastrointestinal ischemia reperfusion damage after CPB.

结论CPB术后的胃粘膜出现缺血再灌注损伤.

Effect of Single Lung Ischemia Reperfusion Injury on Contralateral Lung in Rats

单肺缺血再灌注对对侧肺的影响

Protective effects of tumor necrosis factor α antibody on liver ischemia reperfusion injury in rats

肿瘤坏死因子α抗体对大鼠肝脏缺血再灌注损伤的保护作用

Conclusion Hyperoxia liquid preconditioning may attenuate myocardial ischemia reperfusion injury.

结论高氧液预处理能减轻心肌缺血-再灌注损伤后酶学改变。

Ischemia reperfusion group layer 4 organizational structure variable density, interstitial edema.

缺血再灌注损伤组4层组织结构变致密、间质明显水肿。

Objective To study the level of nitric NOS in ischemia-reperfusion rat liver.

目的探讨大鼠缺血再灌注肝一氧化氮合酶的变化及其意义.

Conclusions Reduced glutathione could protect lung ischemia-reperfusion injury.

结论还原型谷胱甘肽对肺缺血再灌注损伤有保护作用.

The molecular mechanism of protective effect of puerarin on cerebral ischemia reperfusion injury in rats

葛根素对缺血再灌注脑保护作用的可能分子机制

Objective: Hepatic ischemia reperfusion injury can induces intrahepatic cholestasis.

目的: 肝缺血再灌注损伤可造成肝内胆汁淤积.

Methods: The animals achieved hypercapnic acidosis with mechanical hypoventilation before ischemia-reperfusion.

方法在缺血再灌注前,用低潮气量,低频率的机械通气方法造成动物的呼吸性酸中毒.

Impact of Aortic Bypass on Viscera Ischemia reperfusion Injury

主动脉转流对内脏缺血再灌注损伤的影响

The activation of Caspase-3 contributed to myocardial cell apoptosis during ischemia reperfusion.

Caspase-3参与了缺血再灌注导致的心肌细胞凋亡过程，且心肌细胞凋亡是缺血再灌注损伤所致细胞死亡的重要组成部分。

Changes of IL-1 and Leukocyte Infiltration in Model of Cerebral Ischemia Reperfusion of Rats

大鼠局灶脑缺血和（或）再灌流模型中IL-1β的表达及其与白细胞浸润的关系

Objective To study bacterial translocation during mesenteric ischemia reperfusion injury.

目的了解新生儿肠缺血再灌注损伤对细菌移位的影响.

Protective effect of Red Sage Root on lung injury induced by brain ischemia ? reperfusion.

丹参对 脑缺血 再灌注后所致肺损伤的保护作用.

Effect and Mechanism of Glutathione on the Rat Liver Ischemia Reperfusion Injury

还原型谷胱甘肽对大鼠肝脏缺血再灌注损伤的影响及机制探讨

ICAM-1 may play important roles in myocardial ischemia-reperfusion injury after CPB.

ICAM -1 在CPB后心肌缺血再灌注损伤中起重要作用.

The Effect of Acupuncture on Peripheral Blood WBC and Cell Factors in Cerebral Ischemia Reperfusion

针刺对全脑缺血再灌注大鼠外周血WBC和细胞因子的影响

The Elementary Research on Hepatic Carcinoma Following Ischemia Reperfusion Injury

肝癌缺血再灌注损伤的基础研究

Apoptosis is an important pathophysiological characteristic during hepatic ischemia-reperfusion injury ( HIRI ) .

细胞凋亡是肝缺血 — 再 灌注 损伤重要的病理生理特征.

The IPC 、 PMA protected the myocardium from sustained ischemia-reperfusion injury . Bre.

Bre. 则可消除IPC对心肌的保护作用.

Conclusion During ischemia reperfusion injury, the Ca 2+ distribution inside immature myocardial cells correlates with the changes of myocardial ultrastructures.

结论缺血再灌注期间，未成熟心肌细胞内Ca2+分布异常与超微结构的改变有密切关系。

Conclusion: F 2 can antagonize the arrhythmias induced by ischemia-reperfusion in rat heart.

结论: F2对大鼠心脏缺血再灌注心律失常具有拮抗作用.

Myocardial Stunning Model Prepared by Ischemia Reperfusion in Rat

大鼠单次完全可逆缺血/再灌注心肌顿抑模型的改进

ASI can lower expression of NF-kB and decrease cerebral ischemia-reperfusion injury.

刺五加注射液可下调的NF-kB表达,减轻脑缺血再灌注损伤.

Methods A model of retinal ischemia reperfusion in rat was made.

方法制备大鼠视网膜缺血再灌注模型.

Changes of endoplasmic reticulum activity in hippocampus cells after ischemia reperfusion simulated in vitro

体外模拟缺血再灌注后海马细胞内质网活性变化

CONCLUSION DK can protect myocardium against ischemia reperfusion injury.

结论dk对大鼠心肌缺血再灌注损伤有保护作用.